Mice lacking core 1-derived O-glycan in podocytes develop transient proteinuria, resulting in focal segmental glomerulosclerosis-Reference-Cited by-同舟云学术

Mice lacking core 1-derived O-glycan in podocytes develop transient proteinuria, resulting in focal segmental glomerulosclerosis

Published:2020-03 Issue:4 Volume:523 Page:1007-1013
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Fuseya Sayaka,Suzuki Riku,Okada Risa,Hagiwara Kozue,Sato Takashi,Narimatsu Hisashi,Yokoi Hideki,Kasahara Masato,Usui Toshiaki,Morito Naoki,Yamagata Kunihiro,Kudo Takashi,Takahashi Satoru

Funder

New Energy and Industrial Technology Development Organization

JSPS KAKENHI

AMED

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference27 articles.

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2. Defective angiogenesis and fatal embryonic hemorrhage in mice lacking core 1-derived O-glycans;Xia;J. Cell Biol.,2004

3. Platelet biogenesis and functions require correct protein O-glycosylation;Wang;Proc. Natl. Acad. Sci.,2012

4. Thrombocytopenia and kidney disease in mice with a mutation in the C1galt1 gene;Alexander;Proc. Natl. Acad. Sci.,2006

5. C1galt1-deficient mice exhibit thrombocytopenia due to abnormal terminal differentiation of megakaryocytes;Kudo;Blood,2013

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