CARD19, the protein formerly known as BinCARD, is a mitochondrial protein that does not regulate Bcl10-dependent NF-κB activation after TCR engagement-Reference-Cited by-同舟云学术

CARD19, the protein formerly known as BinCARD, is a mitochondrial protein that does not regulate Bcl10-dependent NF-κB activation after TCR engagement

Published:2020-10 Issue: Volume:356 Page:104179
ISSN:0008-8749
Container-title:Cellular Immunology
language:en
Short-container-title:Cellular Immunology

Author:

Rios Kariana E.,Kashyap Anuj K.,Maynard Sean K.,Washington Michael,Paul Suman,Schaefer Brian C.

Funder

National Institute of Allergy and Infectious Diseases

National Institute of General Medical Sciences

Publisher

Elsevier BV

Subject

Immunology

Reference24 articles.

1. CARD games in apoptosis and immunity;Bouchier-Hayes;EMBO Rep.,2002

2. Cutting Edge: TCR Ligation Triggers Digital Activation of NF-κB;Kingeter;J. Immunol.,2010

3. Antigen receptor signaling to NF-κB via CARMA1, BCL10, and MALT1;Thome;Cold Spring Harbor Perspect. Biol.,2010

4. The TRAF6 ubiquitin ligase and TAK1 kinase mediate IKK activation by BCL10 and MALT1 in T lymphocytes;Sun;Mol. Cell,2004

5. Phosphorylation and ubiquitination of the IκB kinase complex by two distinct signaling pathways;Shambharkar;EMBO J.,2007

Cited by 5 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. CARD19, a Novel Regulator of the TAK1/NF-κB Pathway in Self-Reactive B Cells;The Journal of Immunology;2023-03-24

2. CARD19 Interacts with Mitochondrial Contact Site and Cristae Organizing System Constituent Proteins and Regulates Cristae Morphology;Cells;2022-03-31

3. Genetic targeting of Card19 is linked to disrupted NINJ1 expression, impaired cell lysis, and increased susceptibility to Yersinia infection;PLOS Pathogens;2021-10-14

4. Genetic targeting of Card19 is linked to disrupted Ninj1 expression, impaired cell lysis, and increased susceptibility to Yersinia infection;2021-03-20

5. The CBM complex: A growing multiplicity of cellular functions, regulatory mechanisms and connections to human disease;Cellular Immunology;2020-10