JAK/STAT3 Signaling Activation Related to Distinct Clinicopathologic Features in Systemic ALK− Anaplastic Large Cell Lymphomas-Reference-Cited by-同舟云学术

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JAK/STAT3 Signaling Activation Related to Distinct Clinicopathologic Features in Systemic ALK− Anaplastic Large Cell Lymphomas

Published:2022-11-01 Issue:1 Volume:47 Page:55-64
ISSN:0147-5185
Container-title:American Journal of Surgical Pathology
language:en
Short-container-title:

Author:

Wang Jian-chao,Zhong Li-hua,Lin Wei-qing,Zhang Wen-fang,Xi Yan-feng,Liu Yue-ping,Zhu Qiong,Liu Wei,Zhu Wei-feng,Chen Yan-ping,Chen Gang

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Pathology and Forensic Medicine,Surgery,Anatomy

Reference24 articles.

1. ALK-negative anaplastic large cell lymphoma is a genetically heterogeneous disease with widely disparate clinical outcomes;Parrilla Castellar;Blood,2014

2. Identification of a new subclass of ALK-negative ALCL expressing aberrant levels of ERBB4 transcripts;Scarfò;Blood,2016

3. Anaplastic lymphoma kinase (ALK) activates Stat3 and protects hematopoietic cells from cell death;Zamo;Oncogene,2002

4. Stat3 is required for ALK-mediated lymphomagenesis and provides a possible therapeutic target;Chiarle;Nat Med,2005

5. Convergent mutations and kinase fusions lead to oncogenic STAT3 activation in anaplastic large cell lymphoma;Crescenzo;Cancer Cell,2015

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1. DUSP22 Ameliorates Endothelial-to-Mesenchymal Transition in HUVECs through Smad2/3 and MAPK Signaling Pathways;Cardiovascular Therapeutics;2024-03-08

2. Anaplastic large cell lymphomas with equivocal DUSP22 FISH results: recommendations for clinical reporting and diagnostic evaluation;Human Pathology;2023-11

3. How molecular advances may improve the diagnosis and management of PTCL patients;Frontiers in Oncology;2023-06-23

4. Phosphorylated STAT3 as a potential diagnostic and predictive biomarker in ALK- ALCL vs. CD30high PTCL, NOS;Frontiers in Immunology;2023-06-14

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