Two mechanisms of chromosome fragility at replication-termination sites in bacteria-Reference-Cited by-同舟云学术

Two mechanisms of chromosome fragility at replication-termination sites in bacteria

Published:2021-06-18 Issue:25 Volume:7 Page:
ISSN:2375-2548
Container-title:Science Advances
language:en
Short-container-title:Sci. Adv.

Author:

Mei Qian¹²³⁴⁵^ORCID,Fitzgerald Devon M.¹²³⁴^ORCID,Liu Jingjing¹²³⁴,Xia Jun¹²³⁴,Pribis John P.¹²³⁴^ORCID,Zhai Yin¹²³⁴,Nehring Ralf B.¹²³⁴^ORCID,Paiano Jacob⁶^ORCID,Li Heyuan²,Nussenzweig Andre⁶,Hastings P.J.¹⁴,Rosenberg Susan M.¹²³⁴⁵^ORCID

Affiliation:

1. Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX 77030, USA.

2. Department of Biochemistry and Molecular Biology, Baylor College of Medicine, Houston, TX 77030, USA.

3. Department of Molecular Virology and Microbiology, Baylor College of Medicine, Houston, TX 77030, USA.

4. Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX, USA.

5. Systems, Synthetic and Physical Biology Program, Rice University, Houston, TX 77030, USA.

6. Laboratory of Genome Integrity, National Cancer Institute, NIH, Bethesda, MD 20892, USA.

Abstract

Caught in the act, DNA reaction intermediates in bacterial genomes may illuminate disease-driving regions in human chromosomes.

Funder

National Institutes of Health

American Cancer Society

National Cancer Institute

National Institute of General Medical Sciences

W.M. Keck Foundation

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference78 articles.

1. DNA polymerase ? inhibition by aphidicolin induces gaps and breaks at common fragile sites in human chromosomes

2. Fragile sites in cancer: more than meets the eye

3. Human DNA Repair Genes

4. Recombinational Repair of DNA Damage in Escherichia coli and Bacteriophage λ

5. Holliday junction trap shows how cells use recombination and a junction-guardian role of RecQ helicase

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