Abnormal Mitochondrial L-Arginine Transport Contributes to the Pathogenesis of Heart Failure and Rexoygenation Injury-Reference-Cited by-同舟云学术

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Abnormal Mitochondrial L-Arginine Transport Contributes to the Pathogenesis of Heart Failure and Rexoygenation Injury

Published:2014-08-11 Issue:8 Volume:9 Page:e104643
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

Williams David,Venardos Kylie M.,Byrne Melissa,Joshi Mandar,Horlock Duncan,Lam Nicholas T.,Gregorevic Paul,McGee Sean L.,Kaye David M.

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference48 articles.

1. Myocardial ischemia-reperfusion injury, antioxidant enzyme systems, and selenium: a review;KM Venardos;Curr Med Chem,2007

2. Mitochondrial energy metabolism in heart failure: a question of balance;JM Huss;J Clin Invest,2005

3. Mitochondrial electron transport complex I is a potential source of oxygen free radicals in the failing myocardium;T Ide;Circ Res,1999

4. Mitochondrial oxidative stress in heart failure: “oxygen wastage” revisited;DB Sawyer;Circ Res,2000

5. Cytoprotective effects of nitrite during in vivo ischemia-reperfusion of the heart and liver;MR Duranski;Journal of Clinical Investigation,2005

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