Phosphorylation of the Leukemic Oncoprotein EVI1 on Serine 196 Modulates DNA Binding, Transcriptional Repression and Transforming Ability-Reference-Cited by-同舟云学术

Phosphorylation of the Leukemic Oncoprotein EVI1 on Serine 196 Modulates DNA Binding, Transcriptional Repression and Transforming Ability

Published:2013-06-12 Issue:6 Volume:8 Page:e66510
ISSN:1932-6203
Container-title:PLoS ONE
language:en
Short-container-title:PLoS ONE

Author:

White Daniel J.,Unwin Richard D.,Bindels Eric,Pierce Andrew,Teng Hsiang-Ying,Muter Joanne,Greystoke Brigit,Somerville Tim D.,Griffiths John,Lovell Simon,Somervaille Tim C. P.,Delwel Ruud,Whetton Anthony D.,Meyer Stefan

Publisher

Public Library of Science (PLoS)

Subject

Multidisciplinary

Reference49 articles.

1. Evi-1 is a critical regulator for hematopoietic stem cells and transformed leukemic cells;S Goyama;Cell Stem Cell,2008

2. High EVI1 expression predicts poor survival in acute myeloid leukemia: a study of 319 de novo AML patients;S Barjesteh van Waalwijk van Doorn-Khosrovani;Blood,2003

3. High EVI1 levels predict adverse outcome in acute myeloid leukemia: prevalence of EVI1 overexpression and chromosome 3q26 abnormalities underestimated;S Lugthart;Blood,2008

4. Fanconi anemia (FA)-associated 3q gains in leukemic transformation consistently target EVI1, but do not affect low TERC expression in FA;S Meyer;Blood,2011

5. Amplification and translocation of 3q26 with overexpression of EVI1 in Fanconi anemia-derived childhood acute myeloid leukemia with biallelic FANCD1/BRCA2 disruption;S Meyer;Genes Chromosomes Cancer,2007

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