The Role of Endoplasmic Reticulum Stress and NLRP3 Inflammasomes in the Development of Atherosclerosis
Author:
Publisher
Allerton Press
Subject
Genetics,Agricultural and Biological Sciences (miscellaneous),Cell Biology
Link
https://link.springer.com/content/pdf/10.3103/S0095452721040113.pdf
Reference62 articles.
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3. Chai, T.F., Hong, S.Y., He, H., et al., A potential mechanism of metformin-mediated regulation of glucose homeostasis: inhibition of thioredoxin-interacting protein (TXNIP) gene expression, Cell Signal., 2012, vol. 24, pp. 1700–1705.https://doi.org/10.1016/j.cellsig.2012.04.017
4. Cheang, W.S., Tian, X.Y., Wong, W.T., et al., Metformin protects endothelial function in diet-induced obese mice by inhibition of endoplasmic reticulum stress through 5' adenosine monophosphate-activated protein kinase-peroxisome proliferator-activated receptor delta pathway, Arterioscler. Thromb. Vase Biol., 2014, vol. 34, no. 4, pp. 830–836. https://doi.org/10.1161/ATVBAHA.113.301938
5. Chen, X., Guo, X., Ge, Q., et al., ER stress activates the NLRP3 inflammasome: a novel mechanism of atherosclerosis, Oxid. Med. Cell Longev., 2019a, p. 3462530. https://doi.org/10.1155/2019/3462530
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