Reduction in <sup>99m</sup>Tc-DPD myocardial uptake with therapy of ATTR cardiomyopathy-Reference-Cited by-同舟云学术

Reduction in ^99mTc-DPD myocardial uptake with therapy of ATTR cardiomyopathy

Published:2023-08-20 Issue: Volume: Page:1-10
ISSN:1350-6129
Container-title:Amyloid
language:en
Short-container-title:Amyloid

Author:

Rettl René¹,Calabretta Raffaella²,Duca Franz¹,Binder Christina¹,Kronberger Christina¹,Willixhofer Robin¹,Poledniczek Michael¹,Donà Carolina¹,Nitsche Christian¹,Beitzke Dietrich³,Loewe Christian³,Auer-Grumbach Michaela⁴,Bonderman Diana⁵,Kastl Stefan¹,Hengstenberg Christian¹,Badr Eslam Roza¹,Kastner Johannes¹,Bergler-Klein Jutta¹,Hacker Marcus²,Kammerlander Andreas¹

Affiliation:

1. Division of Cardiology, Department of Internal Medicine II, Medical University of Vienna, Vienna, Austria

2. Division of Nuclear Medicine, Department of Biomedical Imaging and Image-guided Therapy, Medical University of Vienna, Vienna, Austria

3. Division of Cardiovascular and Interventional Radiology, Department of Biomedical Imaging and Image-guided Therapy, Medical University of Vienna, Vienna, Austria

4. Department of Orthopaedics and Trauma Surgery, Medical University of Vienna, Vienna, Austria

5. Division of Cardiology, Department of Internal Medicine V, Favoriten Clinic, Vienna, Austria

Publisher

Informa UK Limited

Subject

Internal Medicine

Link

https://www.tandfonline.com/doi/pdf/10.1080/13506129.2023.2247136

Reference37 articles.

1. Hereditary transthyretin amyloidosis: a model of medical progress for a fatal disease

2. Prevalence and clinical phenotype of hereditary transthyretin amyloid cardiomyopathy in patients with increased left ventricular wall thickness

3. Hereditary ATTR Amyloidosis in Austria: Prevalence and Epidemiological Hot Spots