The trace amine theory of spontaneous hypertension as induced by classic monoamine oxidase inhibitors
Author:
Publisher
Springer Science and Business Media LLC
Subject
Biological Psychiatry,Psychiatry and Mental health,Clinical Neurology,Neurology
Link
https://link.springer.com/content/pdf/10.1007/s00702-021-02399-9.pdf
Reference156 articles.
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2. Andersen G, Marcinek P, Sulzinger N, Schieberle P, Krautwurst D (2019) Food sources and biomolecular targets of tyramine. Nutr Rev 77(2):107–115. https://doi.org/10.1093/nutrit/nuy036
3. Baker GB, Coutts RT, Legatt DF (1983) Levels of p-tyramine in rat brain after chronic administration of MAO-inhibiting antidepressants. Prog Neuropsychopharmacol Biol Psychiatry 7:779–782. https://doi.org/10.1016/0278-5846(83)90065-9
4. Baker GB, Legatt DF, Coutts RT (1984) A comparison of effects of acute and chronic administration of phenelzine and tranylcypromine on brain concentrations of 2-phenylethylamine, p-tyramine, and tryptamine in the rat. In: Boulton AA, Baker GB, Dewhurst WG, Sandler M (eds) Neurobiology of the trace amines. The Humana Press Inc., Clifton, pp 277–282
5. Baker GB, Coutts RT, Yeung JM, Hampson DR, McIntosh GJA, McIntosh MG (1985) Chronic administration of monoamine oxidase inhibitors: basic and clinical investigations. In: Boulton AA, Maitre L, Bieck PR, Riederer P (eds) Neuropsychopharmacology of the Trace Amines, pp 317–328. https://doi.org/10.1007/978-1-4612-5010-4_34
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