Septic cardiomyopathy in rat LPS-induced endotoxemia: relative contribution of cellular diastolic Ca2+ removal pathways, myofibrillar biomechanics properties and action of the cardiotonic drug levosimendan-Reference-Cited by-同舟云学术

Septic cardiomyopathy in rat LPS-induced endotoxemia: relative contribution of cellular diastolic Ca2+ removal pathways, myofibrillar biomechanics properties and action of the cardiotonic drug levosimendan

Published:2015-08-05 Issue:5 Volume:110 Page:
ISSN:0300-8428
Container-title:Basic Research in Cardiology
language:en
Short-container-title:Basic Res Cardiol

Author:

Wagner S.,Schürmann S.,Hein S.,Schüttler J.,Friedrich O.

Funder

ELAN Fonds Medical Faculty Erlangen

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://link.springer.com/content/pdf/10.1007/s00395-015-0507-4.pdf

Reference41 articles.

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4. Ceylan-Isik AF, Zhao P, Zhang B, Xiao X, Su G, Ren J (2010) Cardiac overexpression of metallothionein rescues cardiac contractile dysfunction and endoplasmic reticulum stress but not autophagy in sepsis. J Mol Cell Cardiol 48(2):367–378. doi: 10.1016/j.yjmcc.2009.11.003

5. Duncan DJ, Yang Z, Hopkins PM, Steele DS, Harrison SM (2010) TNF-alpha and IL-1beta increase Ca2 + leak from the sarcoplasmic reticulum and susceptibility arrhythmia in rat ventricular myocytes. Cell Calcium 47(4):378–386. doi: 10.1016/j.ceca.2010.02.002

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