Levosimendan Reverses Cardiac Malfunction and Cardiomyocyte Ferroptosis During Heart Failure with Preserved Ejection Fraction via Connexin 43 Signaling Activation-Reference-Cited by-同舟云学术

Levosimendan Reverses Cardiac Malfunction and Cardiomyocyte Ferroptosis During Heart Failure with Preserved Ejection Fraction via Connexin 43 Signaling Activation

Published:2023-03-07 Issue: Volume: Page:
ISSN:0920-3206
Container-title:Cardiovascular Drugs and Therapy
language:en
Short-container-title:Cardiovasc Drugs Ther

Author:

Zhang Li-li,Chen Gui-hao,Tang Rui-jie,Xiong Yu-yan,Pan Qi,Jiang Wen-yang,Gong Zhao-ting,Chen Cheng,Li Xiao-song,Yang Yue-jin^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Pharmacology (medical),Cardiology and Cardiovascular Medicine,Pharmacology,General Medicine

Link

https://link.springer.com/content/pdf/10.1007/s10557-023-07441-4.pdf

Reference50 articles.

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2. Savarese G, Vedin O, D'Amario D, et al. Prevalence and prognostic implications of longitudinal ejection fraction change in heart failure. JACC Heart Fail. 2019;7(4):306–17.

3. Cuijpers I, Simmonds SJ, van Bilsen M, et al. Microvascular and lymphatic dysfunction in HFpEF and its associated comorbidities. Basic Res Cardiol. 2020;115(4):39.

4. Boengler K, Leybaert L, Ruiz-Meana M, Schulz R. Connexin 43 in mitochondria: what do we really know about its function? Front Physiol. 2022;13:928934.

5. Shin KT, Nie ZW, Zhou W, et al. Connexin 43 knockdown induces mitochondrial dysfunction and affects early developmental competence in porcine embryos. Microsc Microanal. 2020;26(2):287–96.

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