SILENCING M6A READER YTHDC1 REDUCES INFLAMMATORY RESPONSE IN SEPSIS-INDUCED CARDIOMYOPATHY BY INHIBITING SERPINA3N EXPRESSION-Reference-Cited by-同舟云学术

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SILENCING M6A READER YTHDC1 REDUCES INFLAMMATORY RESPONSE IN SEPSIS-INDUCED CARDIOMYOPATHY BY INHIBITING SERPINA3N EXPRESSION

Published:2023-03-07 Issue:5 Volume:59 Page:791-802
ISSN:1073-2322
Container-title:Shock
language:en
Short-container-title:Shock

Author:

Xie Wenjing,Zhang Anqi,Huang Xuliang,Zhou Hui,Ying Hangbo,Ye Changzhou,Ren Miao,Qian Meizi,Liu Xia,Mo Yunchang

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Critical Care and Intensive Care Medicine,Emergency Medicine

Reference50 articles.

1. Interleukin-18 gene deletion protects against sepsis-induced cardiac dysfunction by inhibiting PP2A activity;Int J Cardiol,2017

2. Administration of anti-TNF antibody improves left ventricular function in septic shock patients. Results of a pilot study;Chest,1992

3. Oxidative stress and mitochondrial dysfunction in sepsis;Br J Anaesth,2011

4. Sepsis-induced myocardial dysfunction: pathophysiology and management;J Intensive Care,2016

5. Endotoxemia is associated with partial uncoupling of cardiac pacemaker from cholinergic neural control in rats;Shock,2012

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4. YTHDC1 inhibits autophagy-dependent NF-κB signaling by stabilizing Beclin1 mRNA in macrophages;Journal of Inflammation;2024-06-14

5. Regulation of CNS pathology by Serpina3n/SERPINA3: The knowns and the puzzles;Neuropathology and Applied Neurobiology;2024-04

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