Superoxide Dismutases SOD1 and SOD2 Rescue the Toxic Effect of Dopamine-Derived Products in Human SH-SY5Y Neuroblastoma Cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Toxicology,General Neuroscience
Link
http://link.springer.com/content/pdf/10.1007/s12640-019-00078-y.pdf
Reference38 articles.
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2. Bender A, Krishnan KJ, Morris CM, Taylor GA, Reeve AK, Perry RH, Jaros E, Hersheson JS, Betts J, Klopstock T, Taylor RW, Turnbull DM (2006) High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease. Nat Genet 38:515–517. https://doi.org/10.1038/ng1769
3. Berman SB, Hastings TG (1999) Dopamine oxidation alters mitochondrial respiration and induces permeability transition in brain mitochondria: implications for Parkinson’s disease. J Neurochem 73:1127–1137
4. Biosa A, Arduini I, Soriano ME, Giorgio V, Bernardi P, Bisaglia M, Bubacco L (2018a) Dopamine oxidation products as mitochondrial endotoxins, a potential molecular mechanism for preferential neurodegeneration in Parkinson’s disease. ACS Chem Neurosci 9:2849–2858. https://doi.org/10.1021/acschemneuro.8b00276
5. Biosa A, Sanchez-Martinez A, Filograna R, Terriente-Felix A, Alam SM, Beltramini M, Bubacco L, Bisaglia M, Whitworth AJ (2018b) Superoxide dismutating molecules rescue the toxic effects of PINK1 and parkin loss. Hum Mol Genet 27:1618–1629. https://doi.org/10.1093/hmg/ddy069
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