Dopamine Oxidation Products as Mitochondrial Endotoxins, a Potential Molecular Mechanism for Preferential Neurodegeneration in Parkinson’s Disease
Author:
Affiliation:
1. Department of Biology, University of Padova, 35121 Padova, Italy
2. Department of Biomedical Sciences and CNR Neuroscience Institute, University of Padova, 35121 Padova, Italy
Funder
Ministero dell?Istruzione, dell?Universit? e della Ricerca
Publisher
American Chemical Society (ACS)
Subject
Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine
Link
https://pubs.acs.org/doi/pdf/10.1021/acschemneuro.8b00276
Reference45 articles.
1. Dopaminergic neurons
2. Dysfunction of dopamine homeostasis: clues in the hunt for novel Parkinson's disease therapies
3. Neuropathology of Parkinsonʼs Disease
4. Mitochondrial Complex I Deficiency in Parkinson's Disease
5. Iron and Aluminum Increase in the Substantia Nigra of Patients with Parkinson's Disease: An X-Ray Microanalysis
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