Superoxide dismutating molecules rescue the toxic effects of PINK1 and parkin loss
Author:
Affiliation:
1. Molecular Physiology and Biophysics Unit, Department of Biology, University of Padova, 35131 Padova, Italy
2. MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge Biomedical Campus, Cambridge CB2 0XY, UK
Funder
ERC
Medical Research Council
Publisher
Oxford University Press (OUP)
Subject
Genetics (clinical),Genetics,Molecular Biology,General Medicine
Link
http://academic.oup.com/hmg/article-pdf/27/9/1618/25087288/ddy069.pdf
Reference49 articles.
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3. Redox homeostasis and mitochondrial dynamics;Willems;Cell Metab,2015
4. Genetic etiology of Parkinson disease associated with mutations in the SNCA, PARK2, PINK1, PARK7, and LRRK2 genes: a mutation update;Nuytemans;Hum. Mutat,2010
5. Mitochondrial dysfunction and oxidative stress in Parkinson's disease and monogenic parkinsonism;Hauser;Neurobiol. Dis,2013
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