Superoxide dismutating molecules rescue the toxic effects of PINK1 and parkin loss
Author:
Affiliation:
1. Molecular Physiology and Biophysics Unit, Department of Biology, University of Padova, 35131 Padova, Italy
2. MRC Mitochondrial Biology Unit, University of Cambridge, Cambridge Biomedical Campus, Cambridge CB2 0XY, UK
Funder
ERC
Medical Research Council
Publisher
Oxford University Press (OUP)
Subject
Genetics (clinical),Genetics,Molecular Biology,General Medicine
Link
http://academic.oup.com/hmg/article-pdf/27/9/1618/25087288/ddy069.pdf
Reference49 articles.
1. Mitochondrial dysfunction and oxidative stress in Parkinson's disease;Subramaniam;Prog. Neurobiol,2013
2. Mitochondrial fusion and fission in cell life and death;Westermann;Nat. Rev. Mol. Cell. Biol,2010
3. Redox homeostasis and mitochondrial dynamics;Willems;Cell Metab,2015
4. Genetic etiology of Parkinson disease associated with mutations in the SNCA, PARK2, PINK1, PARK7, and LRRK2 genes: a mutation update;Nuytemans;Hum. Mutat,2010
5. Mitochondrial dysfunction and oxidative stress in Parkinson's disease and monogenic parkinsonism;Hauser;Neurobiol. Dis,2013
Cited by 28 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Elevated Level of PINK1/Parkin-Mediated Mitophagy Pathway Involved to the Inhibited Activity of Mitochondrial Superoxide Dismutase in Rat Brains and Primary Hippocampal Neurons Exposed to High Level of Fluoride;Biological Trace Element Research;2023-05-16
2. Modeling of Parkinson’s disease in Drosophila based on DJ-1 deficiency;Handbook of Animal Models in Neurological Disorders;2023
3. A water-soluble manganese(II) octanediaoate/phenanthroline complex acts as an antioxidant and attenuates alpha-synuclein toxicity;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2022-10
4. Oxidative Stress Inhibition Via Apocynin Prevents Medullary Respiratory Neurodegeneration and Respiratory Pattern Dysfunction in a 6-Hydroxydopamine Animal Model of Parkinson’s Disease;Neuroscience;2022-10
5. Mitochondrial electron transport chain defects modify Parkinson's disease phenotypes in a Drosophila model;Neurobiology of Disease;2022-09
1.学者识别学者识别
2.学术分析学术分析
3.人才评估人才评估
"同舟云学术"是以全球学者为主线,采集、加工和组织学术论文而形成的新型学术文献查询和分析系统,可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容,当前同舟云学术共收录了国内外主流学术期刊6万余种,收集的期刊论文及会议论文总量共计约1.5亿篇,并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询!咨询电话:010-8811{复制后删除}0370
www.globalauthorid.com
TOP
Copyright © 2019-2024 北京同舟云网络信息技术有限公司 京公网安备11010802033243号 京ICP备18003416号-3