AMPK/mTOR/p70S6K axis prevents apoptosis of Porphyromonas gingivalis-infected gingival epithelial cells via BadSer136 phosphorylation-Reference-Cited by-同舟云学术

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AMPK/mTOR/p70S6K axis prevents apoptosis of Porphyromonas gingivalis-infected gingival epithelial cells via BadSer136 phosphorylation

Published:2023-04-04 Issue:7-8 Volume:28 Page:1012-1023
ISSN:1360-8185
Container-title:Apoptosis
language:en
Short-container-title:Apoptosis

Author:

Wang Yanchun^ORCID,Dong Yilong^ORCID,Zhang Wenbo^ORCID,Wang Yanmei^ORCID,Jao Yang^ORCID,Liu Jianjun^ORCID,Zhang Mingzhu^ORCID,He Hongbing^ORCID

Funder

the Joint Special Funds for the Department of Science and Technology of Yunnan Province-Kunming Medical University

Kunming Medical University PhD Student Innovation Fund

National Nature Science Foundation of China

the high-level health technology talent project in Yunnan Province

Joint Special Funds for the Department of Science and Technology of Yunnan Province-Kunming Medical University

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Biochemistry (medical),Cell Biology,Clinical Biochemistry,Pharmaceutical Science,Pharmacology

Link

https://link.springer.com/content/pdf/10.1007/s10495-023-01839-z.pdf

Reference60 articles.

1. Hajishengallis G, Darveau RP, Curtis MA (2012) The keystone-pathogen hypothesis. Nat Rev Microbiol 10:717–725. https://doi.org/10.1038/nrmicro2873

2. Hajishengallis G, Liang S, Payne MA et al (2011) Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement. Cell Host Microbe 10:497–506. https://doi.org/10.1016/j.chom.2011.10.006

3. Hajishengallis G, Lambris JD (2011) Microbial manipulation of receptor crosstalk in innate immunity. Nat Rev Immunol 11:187–200. https://doi.org/10.1038/nri2918

4. Wang M, Krauss JL, Domon H, et al. Microbial hijacking of complement-toll-like receptor crosstalk. Sci Signal 2010;3:ra11. doi:https://doi.org/10.1126/scisignal.2000697.

5. Maekawa T, Krauss JL, Abe T et al (2014) Porphyromonas gingivalis manipulates complement and TLR signaling to uncouple bacterial clearance from inflammation and promote dysbiosis. Cell Host Microbe 15:768–778. https://doi.org/10.1016/j.chom.2014.05.012

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