Microbial Hijacking of Complement–Toll-Like Receptor Crosstalk-Reference-Cited by-同舟云学术

Microbial Hijacking of Complement–Toll-Like Receptor Crosstalk

Published:2010-02-16 Issue:109 Volume:3 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Wang Min¹,Krauss Jennifer L.¹²,Domon Hisanori²,Hosur Kavita B.²,Liang Shuang²,Magotti Paola³,Triantafilou Martha⁴,Triantafilou Kathy⁴,Lambris John D.³,Hajishengallis George¹²

Affiliation:

1. Department of Microbiology and Immunology, University of Louisville School of Medicine, Louisville, KY 40292, USA.

2. Oral Health and Systemic Disease, University of Louisville School of Dentistry, Louisville, KY 40292, USA.

3. Department of Pathology and Laboratory Medicine, University of Pennsylvania School of Medicine, Philadelphia, PA 19104, USA.

4. Infection and Immunity Group, University of Sussex School of Life Sciences, Falmer, Brighton, BN1 9QG, UK.

Abstract

Pathogen-instigated crosstalk between the complement C5a receptor and Toll-like receptor 2 disables innate immune function.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference53 articles.

1. The Role of Complement in Inflammatory Diseases From Behind the Scenes into the Spotlight

2. C5a Negatively Regulates Toll-like Receptor 4-Induced Immune Responses

3. Regulation of Toll-like receptor–mediated inflammatory response by complement in vivo

4. Crosstalk pathways between Toll-like receptors and the complement system.;Hajishengallis G.;Trends Immunol.

5. TLRs and innate immunity

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