Porphyromonas gingivalis Manipulates Complement and TLR Signaling to Uncouple Bacterial Clearance from Inflammation and Promote Dysbiosis-Reference-Cited by-同舟云学术

Porphyromonas gingivalis Manipulates Complement and TLR Signaling to Uncouple Bacterial Clearance from Inflammation and Promote Dysbiosis

Published:2014-06 Issue:6 Volume:15 Page:768-778
ISSN:1931-3128
Container-title:Cell Host & Microbe
language:en
Short-container-title:Cell Host & Microbe

Author:

Maekawa Tomoki,Krauss Jennifer L.,Abe Toshiharu,Jotwani Ravi,Triantafilou Martha,Triantafilou Kathy,Hashim Ahmed,Hoch Shifra,Curtis Michael A.,Nussbaum Gabriel,Lambris John D.,Hajishengallis George

Funder

National Institutes of Health

Medical Research Council

European Commission

Publisher

Elsevier BV

Subject

Virology,Microbiology,Parasitology

Reference44 articles.

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3. Signalling mechanisms for Toll-like receptor-activated neutrophil exocytosis: key roles for interleukin-1-receptor-associated kinase-4 and phosphatidylinositol 3-kinase but not Toll/IL-1 receptor (TIR) domain-containing adaptor inducing IFN-β (TRIF);Brzezinska;Immunology,2009

4. TLR2-dependent inflammatory response to Porphyromonas gingivalis is MyD88 independent, whereas MyD88 is required to clear infection;Burns;J. Immunol.,2010

5. Porphyromonas gingivalis-nucleoside-diphosphate-kinase inhibits ATP-induced reactive-oxygen-species via P2X7 receptor/NADPH-oxidase signalling and contributes to persistence;Choi;Cell. Microbiol.,2013

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