KCNQ1OT1 inhibition alleviates high glucose-induced podocyte injury by adsorbing miR-23b-3p and regulating Sema3A-Reference-Cited by-同舟云学术

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KCNQ1OT1 inhibition alleviates high glucose-induced podocyte injury by adsorbing miR-23b-3p and regulating Sema3A

Published:2022-01-08 Issue:5 Volume:26 Page:385-397
ISSN:1342-1751
Container-title:Clinical and Experimental Nephrology
language:en
Short-container-title:Clin Exp Nephrol

Author:

Fei Bingru,Zhou Hui,He Zengjiao,Wang Suyu

Funder

Youth Fund Project of Natural Science Foundation of China

Publisher

Springer Science and Business Media LLC

Subject

Physiology (medical),Nephrology,Physiology

Link

https://link.springer.com/content/pdf/10.1007/s10157-021-02173-x.pdf

Reference40 articles.

1. Murphy M, Crean J, Brazil DP, Sadlier D, Martin F, Godson C. Regulation and consequences of differential gene expression in diabetic kidney disease. Biochem Soc Trans. 2008;36(Pt 5):941–5.

2. Vasanth Rao VRA/LB, Tan SH, Candasamy M, Bhattamisra SK. Diabetic nephropathy: an update on pathogenesis and drug development. Diabetes Metab Syndr. 2019;13(1):754–62.

3. Schwarz A, Möller-Hackbarth K, Ebarasi L, Unnersjö Jess D, Zambrano S, Blom H, Wernerson A, Lal M, Patrakka J. Coro2b, a podocyte protein downregulated in human diabetic nephropathy, is involved in the development of protamine sulphate-induced foot process effacement. Sci Rep. 2019;9(1):8888.

4. Wolf G, Chen S, Ziyadeh FN. From the periphery of the glomerular capillary wall toward the center of disease: podocyte injury comes of age in diabetic nephropathy. Diabetes. 2005;54(6):1626–34.

5. Miyauchi M, Toyoda M, Kobayashi K, Abe M, Kobayashi T, Kato M, Yamamoto N, Kimura M, Umezono T, Suzuki D. Hypertrophy and loss of podocytes in diabetic nephropathy. Intern Med. 2009;48(18):1615–20.

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3. Long noncoding RNA protein-disulfide isomerase-associated 3 regulated high glucose-induced podocyte apoptosis in diabetic nephropathy through targeting miR-139-3p;World Journal of Diabetes;2024-02-15

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