The role of Jun transcription factor expression and phosphorylation in neuronal differentiation, neuronal cell death, and plastic adaptationsin vivo

Author:

Schlingensiepen Karl-Hermann,Wollnik Franziska,Kunst Mechthild,Schlingensiepen Reimar,Herdegen Thomas,Brysch Wolfgang

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology,Cellular and Molecular Neuroscience,General Medicine

Reference34 articles.

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2. Binetruy, B., Smeal, T., and Karin, M. (1991). Ha-ras augments c-Jun activity and stimulates phosphorylation of its activation domain.Nature 351122–127.

3. Brysch, W., Rifai, A., Tischmeyer, W., and Schlingensiepen, K.-H. (1994). Rational drug design, pharmacokinetics, intracerebral application and organ uptake of antisense phosphorothioate oligodeoxynucleotides. In (S. Agrawal, Ed.),Antisense Oligonucleotide Therapy: Current Status, Humana Press, Clifton, NJ (in press).

4. Chiu, R., Angel, P., and Karin, M. (1989). Jun-B differs in its biological properties from and is a negative regulator of c-Jun.Cell 59979–986.

5. Daan, S., and Pittendrigh, C. S. (1976). A functional analysis of circadian pacemakers in nocturnal rodents. II. The variability of phase response curves.J. Comp. Physiol. A 106253–266.

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