The Rationale for BAFF Inhibition in Systemic Lupus Erythematosus
Author:
Publisher
Springer Science and Business Media LLC
Subject
Rheumatology
Link
http://link.springer.com/content/pdf/10.1007/s11926-012-0258-2.pdf
Reference44 articles.
1. Mackay F, Figgett WA, Saulep D, et al. B-cell stage and context-dependent requirements for survival signals from BAFF and the B-cell receptor. Immunol Rev. 2010;237:205–25.
2. • Mackay F, Schneider P. Cracking the BAFF code. Nat Rev Immunol. 2009;9:491–502. This is an excellent review about BAFF and APRIL and their receptors.
3. Gross JA, Johnston J, Mudri S, et al. TACI and BCMA are receptors for a TNF homologue implicated in B-cell autoimmune disease. Nature. 2000;404(6781):995–9.
4. Bossen C, Cachero TG, Tardivel A, et al. TACI, unlike BAFF-R, is solely activated by oligomeric BAFF and APRIL to support survival of activated B cells and plasmablasts. Blood. 2008;111:1004–12.
5. Gavin AL, Duong B, Skog P, et al. {Delta}BAFF, a splice isoform of BAFF, opposes full-length baff activity in vivo in transgenic mouse models. J Immunol. 2005;175:319–28.
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