ΔBAFF, a Splice Isoform of BAFF, Opposes Full-Length BAFF Activity In Vivo in Transgenic Mouse Models-Reference-Cited by-同舟云学术

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ΔBAFF, a Splice Isoform of BAFF, Opposes Full-Length BAFF Activity In Vivo in Transgenic Mouse Models

Published:2005-06-21 Issue:1 Volume:175 Page:319-328
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Gavin Amanda L.,Duong Bao,Skog Patrick,Aït-Azzouzene Djemel,Greaves David R.,Scott Martin L.,Nemazee David

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference49 articles.

1. TACI-Ig Neutralizes Molecules Critical for B Cell Development and Autoimmune Disease

2. An Essential Role for BAFF in the Normal Development of B Cells Through a BCMA-Independent Pathway

3. Baff Binds to the Tumor Necrosis Factor Receptor–Like Molecule B Cell Maturation Antigen and Is Important for Maintaining the Peripheral B Cell Population

4. Identification of a novel receptor for B lymphocyte stimulator that is mutated in a mouse strain with severe B cell deficiency

5. Competition for BLyS-mediated signaling through Bcmd/BR3 regulates peripheral B lymphocyte numbers

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1. Telitacicept: A novel horizon in targeting autoimmunity and rheumatic diseases;Journal of Autoimmunity;2024-09

2. Past and present discovery of the BAFF/APRIL system – A bibliometric study from 1999 to 2023;Cellular Signalling;2024-08

3. B Cells in Systemic Lupus Erythematosus;Rheumatic Disease Clinics of North America;2021-08

4. TACI haploinsufficiency protects against BAFF‐driven humoral autoimmunity in mice;European Journal of Immunology;2021-06-27

5. Treatment-Induced BAFF Expression and B Cell Biology in Multiple Sclerosis;Frontiers in Immunology;2021-05-26

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