TRPM2 Channel Inhibition Attenuates Amyloid β42-Induced Apoptosis and Oxidative Stress in the Hippocampus of Mice
Author:
Funder
Süleyman Demirel Üniversitesi
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Cellular and Molecular Neuroscience,General Medicine
Link
https://link.springer.com/content/pdf/10.1007/s10571-022-01253-0.pdf
Reference56 articles.
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2. Abuarab N, Munsey TS, Jiang LH, Li J, Sivaprasadarao A (2017) High glucose-induced ROS activates TRPM2 to trigger lysosomal membrane permeabilization and Zn2+-mediated mitochondrial fission. Sci Signal 10(490):eaal4161. https://doi.org/10.1126/scisignal.aal4161
3. Ai J, Wang H, Chu P, Shopit A, Niu M, Ahmad N et al (2021) The neuroprotective effects of phosphocreatine on Amyloid Beta 25–35-induced differentiated neuronal cell death through inhibition of AKT /GSK-3β /Tau/APP /CDK5 pathways in vivo and vitro. Free Radic Biol Med 162:181–190. https://doi.org/10.1016/j.freeradbiomed.2020.10.003
4. Akpınar O, Özşimşek A, Güzel M, Nazıroğlu M (2020) Clostridium botulinum neurotoxin A induces apoptosis and mitochondrial oxidative stress via activation of TRPM2 channel signaling pathway in neuroblastoma and glioblastoma tumor cells. J Recept Signal Transduct Res 40(6):620–632. https://doi.org/10.1080/10799893.2020.1781174
5. Aksenov MY, Tucker HM, Nair P, Aksenova MV, Butterfield DA, Estus S, Markesbery WR (1998) The expression of key oxidative stress-handling genes in different brain regions in Alzheimer’s disease. J Mol Neurosci 11(2):151–164. https://doi.org/10.1385/JMN:11:2:151
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