Clostridium botulinum neurotoxin A induces apoptosis and mitochondrial oxidative stress via activation of TRPM2 channel signaling pathway in neuroblastoma and glioblastoma tumor cells-Reference-Cited by-同舟云学术

Clostridium botulinum neurotoxin A induces apoptosis and mitochondrial oxidative stress via activation of TRPM2 channel signaling pathway in neuroblastoma and glioblastoma tumor cells

Published:2020-07-09 Issue:6 Volume:40 Page:620-632
ISSN:1079-9893
Container-title:Journal of Receptors and Signal Transduction
language:en
Short-container-title:Journal of Receptors and Signal Transduction

Author:

Akpınar Orhan¹²,Özşimşek Ahmet³,Güzel Mustafa⁴,Nazıroğlu Mustafa⁵⁶^ORCID

Affiliation:

1. Unit of Medical Microbiology, Department of Oral and Maxillofacial Surgery, Faculty of Dentistry, Suleyman Demirel University, Isparta, Turkey

2. Departmant of Medical Microbiology, Health Sciences Institute, Suleyman Demirel University, Isparta, Turkey

3. Department of Neurology, Faculty of Medicine, Alanya Alaaddin Keykubat University, Antalya, Turkey

4. Department of Medical Microbiology, Private Maltepe Medical Center, Istanbul, Turkey

5. Neuroscience Research Center, Suleyman Demirel University, Isparta, Turkey

6. Drug Discovery Unit, BSN Health, Analyses, Innovation, Consultancy, Organization, Agriculture, Industry and Trade Limited Company, Göller Bölgesi Teknokenti, Isparta, Turkey

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://www.tandfonline.com/doi/pdf/10.1080/10799893.2020.1781174

Reference41 articles.

1. Correlation of Nrf2 and HIF-1α in glioblastoma and their relationships to clinicopathologic features and survival

2. Lead Exposure, Polymorphisms in Genes Related to Oxidative Stress, and Risk of Adult Brain Tumors

3. Cancer risk in children and young adults born preterm: A systematic review and meta-analysis

4. Association Between Supratotal Glioblastoma Resection and Patient Survival: A Systematic Review and Meta-Analysis

5. Epigenetics in Brain Tumors: HDACs Take Center Stage

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