TRPV4 facilitates the reprogramming of inflamed macrophages by regulating IL-10 production via CREB-Reference-Cited by-同舟云学术

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TRPV4 facilitates the reprogramming of inflamed macrophages by regulating IL-10 production via CREB

Published:2024-08-05 Issue: Volume: Page:
ISSN:1023-3830
Container-title:Inflammation Research
language:en
Short-container-title:Inflamm. Res.

Author:

Arfath Yassir,Kotra Tusharika,Faizan Md Imam,Akhtar Areej,Abdullah Sheikh Tasduq,Ahmad Tanveer,Ahmed Zabeer,Rayees Sheikh

Funder

UGC-CSIR

CSIR

Jamia Millia Islamia

Department of Biotechnology, Ramalingaswami Fellow grant

Publisher

Springer Science and Business Media LLC

Link

https://link.springer.com/content/pdf/10.1007/s00011-024-01923-3.pdf

Reference31 articles.

1. Alharbi MO, et al. Identification and functional analysis of a biflavone as a novel inhibitor of transient receptor potential vanilloid 4-dependent atherogenic processes. Sci Rep. 2021;11(1):8173.

2. Tsuno N, et al. Pharmacological evaluation of novel (6-aminopyridin-3-yl)(4-(pyridin-2-yl) piperazin-1-yl) methanone derivatives as TRPV4 antagonists for the treatment of pain. Bioorg Med Chem. 2017;25(7):2177–90.

3. Luo J, et al. TRPV4 channel signaling in macrophages promotes gastrointestinal motility via direct effects on smooth muscle cells. Immunity. 2018;49(1):107–19. e4.

4. Joshi JC, et al. SPHK2-generated S1P in CD11b + macrophages blocks STING to suppress the inflammatory function of alveolar macrophages. Cell Rep. 2020;30(12):4096–109. e5.

5. Ip WE, et al. Anti-inflammatory effect of IL-10 mediated by metabolic reprogramming of macrophages. Science. 2017;356(6337):513–9.

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