Anti-inflammatory effect of IL-10 mediated by metabolic reprogramming of macrophages
Author:
Affiliation:
1. Department of Immunobiology and Howard Hughes Medical Institute, Yale University School of Medicine, New Haven, CT 06510, USA.
2. Boston Children’s Hospital, Harvard Medical School, Boston, MA 02115, USA.
Abstract
Funder
National Institutes of Health
Howard Hughes Medical Institute
Leona M. and Harry B. Helmsley Charitable Trust
Else Kröner-Fresenius-Stiftung
Blavatnik Family Foundation
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference39 articles.
1. Regulation and Functions of the IL-10 Family of Cytokines in Inflammation and Disease
2. Interleukin-10 Receptor Signaling in Innate Immune Cells Regulates Mucosal Immune Tolerance and Anti-Inflammatory Macrophage Function
3. Macrophage-Restricted Interleukin-10 Receptor Deficiency, but Not IL-10 Deficiency, Causes Severe Spontaneous Colitis
4. Sequence variants in IL10, ARPC2 and multiple other loci contribute to ulcerative colitis susceptibility
5. Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci
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