Monocytes augment inflammatory responses in human aortic valve interstitial cells via β2-integrin/ICAM-1-mediated signaling
Author:
Funder
National Heart, Lung, and Blood Institute
National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Subject
Pharmacology,Immunology
Link
https://link.springer.com/content/pdf/10.1007/s00011-022-01566-2.pdf
Reference52 articles.
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3. Coté N, Mahmut A, Bosse Y, Couture C, Pagé S, Trahan S, et al. Inflammation is associated with the remodeling of calcific aortic valve disease. Inflammation. 2013;36:573–81. https://doi.org/10.1007/s10753-012-9579-6.
4. García-Rodríguez C, Parra-Izquierdo I, Castaños-Mollor I, López J, San Román JA, Sánchez CM. Toll-like receptors, inflammation, and calcific aortic valve disease. Front Physiol. 2018;9:201. https://doi.org/10.3389/fphys.2018.00201.
5. Hutcheson JD, Aikawa E, Merryman WD. Potential drug targets for calcific aortic valve disease. Nat Rev Cardiol. 2014;11:218–31. https://doi.org/10.1038/nrcardio.2014.1.
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