Analgesia by deletion of spinal neurokinin 1 receptor expressing neurons using a bioengineered substance P-Pseudomonas exotoxin conjugate-Reference-Cited by-同舟云学术

Analgesia by deletion of spinal neurokinin 1 receptor expressing neurons using a bioengineered substance P-Pseudomonas exotoxin conjugate

Published:2017-01 Issue: Volume:13 Page:174480691772765
ISSN:1744-8069
Container-title:Molecular Pain
language:en
Short-container-title:Mol Pain

Author:

Iadarola Michael J¹,Sapio Matthew R¹^ORCID,Wang Xunde²,Carrero Hector³,Virata-Theimer Maria Luisa²,Sarnovsky Robert²,Mannes Andrew J¹,FitzGerald David J²

Affiliation:

1. Department of Perioperative Medicine, Clinical Center, National Institutes of Health, Bethesda, MD, USA

2. Biotherapy Section, Laboratory of Molecular Biology, Center for Cancer Research, National Cancer Institute, Bethesda, MD, USA

3. Pain and Neurosensory Mechanisms Branch, National Institutes of Dental and Craniofacial Research, National Institutes of Health, Bethesda, MD, USA

Publisher

SAGE Publications

Subject

Anesthesiology and Pain Medicine,Cellular and Molecular Neuroscience,Molecular Medicine

Link

http://journals.sagepub.com/doi/pdf/10.1177/1744806917727657

Reference78 articles.

1. Deletion of vanilloid receptor 1_expressing primary afferent neurons for pain control

2. Preclinical Toxicity Evaluation of AAV for Pain: Evidence from Human AAV Studies and from the Pharmacology of Analgesic Drugs

3. Inhibition of Hyperalgesia by Ablation of Lamina I Spinal Neurons Expressing the Substance P Receptor

4. AAV for pain: steps towards clinical translation

5. A Paracrine Paradigm for in Vivo Gene Therapy in the Central Nervous System: Treatment of Chronic Pain

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