Fas-Mediated Apoptosome Formation Is Dependent on Reactive Oxygen Species Derived from Mitochondrial Permeability Transition in Jurkat Cells-Reference-Cited by-同舟云学术

Fas-Mediated Apoptosome Formation Is Dependent on Reactive Oxygen Species Derived from Mitochondrial Permeability Transition in Jurkat Cells

Published:2004-06-21 Issue:1 Volume:173 Page:285-296
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Sato Tsutomu,Machida Takuro,Takahashi Sho,Iyama Satoshi,Sato Yasushi,Kuribayashi Kageaki,Takada Kohichi,Oku Takatomi,Kawano Yutaka,Okamoto Tetsuro,Takimoto Rishu,Matsunaga Takuya,Takayama Tetsuji,Takahashi Minoru,Kato Junji,Niitsu Yoshiro

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference32 articles.

1. Cytotoxicity-dependent APO-1 (Fas/CD95)-associated proteins form a death-inducing signaling complex (DISC) with the receptor.

2. Proteases for Cell Suicide: Functions and Regulation of Caspases

3. Glutathione Levels and Sensitivity to Apoptosis Are Regulated by Changes in Transaldolase Expression

4. Fas-mediated apoptosis is modulated by intracellular glutathione in human T cells

5. Thiol-mediated inhibition of FAS and CD2 apoptotic signaling in activated human peripheral T cells

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