Complete Loss of Fas Ligand Gene Causes Massive Lymphoproliferation and Early Death, Indicating a Residual Activity of gld Allele-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Complete Loss of Fas Ligand Gene Causes Massive Lymphoproliferation and Early Death, Indicating a Residual Activity of gld Allele

Published:2004-02-05 Issue:4 Volume:172 Page:2118-2125
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Karray Saoussen,Kress Chantal,Cuvellier Sylvain,Hue-Beauvais Catherine,Damotte Diane,Babinet Charles,Lévi-Strauss Matthieu

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference34 articles.

1. The Fas Death Factor

2. Lpr and gld: Single Gene Models of Systemic Autoimmunity and Lymphoproliferative Disease

3. Generalized lymphoproliferative disease in mice, caused by a point mutation in the fas ligand

4. Targeted mutation in the Fas gene causes hyperplasia in peripheral lymphoid organs and liver

5. A role for CD95 ligand in preventing graft rejection

Cited by 61 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Fas ligand intracellular signaling: does PSTPIP mediate T cell death?;Apoptosis;2023-10-04

2. The complex web of FasL: cell type–specific roles in affecting and controlling acute graft-vs-host disease;Journal of Leukocyte Biology;2023-07-11

3. Apoptotic cell death in disease—Current understanding of the NCCD 2023;Cell Death & Differentiation;2023-04-26

4. Ambivalent role of FasL in murine acute graft-versus-host-disease;Journal of Leukocyte Biology;2023-04-04

5. Antigen-specific depletion of CD4 ⁺ T cells by CAR T cells reveals distinct roles of higher- and lower-affinity TCRs during autoimmunity;Science Immunology;2022-10-14

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3