The complex web of FasL: cell type–specific roles in affecting and controlling acute graft-vs-host disease-Reference-Cited by-同舟云学术

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The complex web of FasL: cell type–specific roles in affecting and controlling acute graft-vs-host disease

Published:2023-07-11 Issue:3 Volume:114 Page:202-204
ISSN:1938-3673
Container-title:Journal of Leukocyte Biology
language:en
Short-container-title:

Author:

Choy Jonathan C¹

Affiliation:

1. Department of Molecular Biology and Biochemistry, Simon Fraser University , 8888 University Drive, Burnaby, British Columbia V5A 1S6 , Canada

Abstract

Abstract FasL has divergent roles in both causing graft-vs-host disease and preventing this condition, which depends on the immune cell type that expresses it.

Publisher

Oxford University Press (OUP)

Subject

Cell Biology,Immunology,Immunology and Allergy

Link

https://academic.oup.com/jleukbio/advance-article-pdf/doi/10.1093/jleuko/qiad079/50912929/qiad079.pdf

Reference16 articles.

1. The Fas death factor;Nagata;Science,1995

2. NFκb activation by Fas is mediated through FADD, caspase-8, and RIP and is inhibited by FLIP;Kreuz;J Cell Biol,2004

3. Fas ligand-deficient gld mice are more susceptible to graft-versus-host-disease;van den Brink;Transplantation,2000

4. Complete loss of Fas ligand gene causes massive lymphoproliferation and early death, indicating a residual activity of gld allele;Karray;J Immunol,2004

5. Ambivalent role of FasL in murine acute graft-versus-host-disease;Bernard-Bloch;J Leukoc Biol,2023

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