Synovial Fibroblasts Directly Induce Th17 Pathogenicity via the Cyclooxygenase/Prostaglandin E2 Pathway, Independent of IL-23-Reference-Cited by-同舟云学术

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Synovial Fibroblasts Directly Induce Th17 Pathogenicity via the Cyclooxygenase/Prostaglandin E2 Pathway, Independent of IL-23

Published:2013-07-01 Issue:3 Volume:191 Page:1364-1372
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Paulissen Sandra M. J.,van Hamburg Jan Piet,Davelaar Nadine,Asmawidjaja Patrick S.,Hazes Johanna M. W.,Lubberts Erik

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference54 articles.

1. Requirement of Interleukin 17 Receptor Signaling for Lung Cxc Chemokine and Granulocyte Colony-Stimulating Factor Expression, Neutrophil Recruitment, and Host Defense

2. Transforming growth factor-β induces development of the TH17 lineage

3. Requirement of Interleukin‐17A for Systemic Anti–Candida albicansHost Defense in Mice

5. IL-17/IL-17 receptor system in autoimmune disease: mechanisms and therapeutic potential

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