Engineered Pigs Carrying a Gain-of-Function NLRP3 Homozygous Mutation Can Survive to Adulthood and Accurately Recapitulate Human Systemic Spontaneous Inflammatory Responses-Reference-Cited by-同舟云学术

Engineered Pigs Carrying a Gain-of-Function NLRP3 Homozygous Mutation Can Survive to Adulthood and Accurately Recapitulate Human Systemic Spontaneous Inflammatory Responses

Published:2020-09-21 Issue:9 Volume:205 Page:2532-2544
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Li Wenjing,Shi Lei,Zhuang Zhenpeng,Wu Han,Lian Meng,Chen Yihui,Li Lei,Ge Weikai,Jin Qin,Zhang Quanjun,Zhao Yu,Liu Zhaoming,Ouyang Zhen,Ye Yinghua,Li Yingying,Wang Hai,Liao Yuan,Quan Longquan,Xiao Lei,Lai Liangxue,Meng Guangxun^ORCID,Wang Kepin

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference44 articles.

1. The NLR Gene Family: A Standard Nomenclature

2. The inflammasome: a danger sensing complex triggering innate immunity

3. Molecular basis of the spectral expression of CIAS1 mutations associated with phagocytic cell-mediated autoinflammatory disorders CINCA/NOMID, MWS, and FCU

4. The calcium-sensing receptor regulates the NLRP3 inflammasome through Ca2+ and cAMP

5. The clinical continuum of cryopyrinopathies: Novel CIAS1 mutations in North American patients and a new cryopyrin model

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