Cutting Edge: Lymphoproliferation Caused by Fas Deficiency Is Dependent on the Transcription Factor Eomesodermin-Reference-Cited by-同舟云学术

Cutting Edge: Lymphoproliferation Caused by Fas Deficiency Is Dependent on the Transcription Factor Eomesodermin

Published:2010-11-12 Issue:12 Volume:185 Page:7151-7155
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Kinjyo Ichiko,Gordon Scott M.,Intlekofer Andrew M.,Dowdell Kennichi,Mooney Erin C.,Caricchio Roberto,Grupp Stephan A.,Teachey David T.,Rao V. Koneti,Lindsten Tullia,Reiner Steven L.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference24 articles.

1. Revised diagnostic criteria and classification for the autoimmune lymphoproliferative syndrome (ALPS): report from the 2009 NIH International Workshop

2. Identifying autoimmune lymphoproliferative syndrome in children with Evans syndrome: a multi-institutional study

3. Elimination of Antigen-Presenting Cells and Autoreactive T Cells by Fas Contributes to Prevention of Autoimmunity

4. Fas Receptor Expression in Germinal-Center B Cells Is Essential for T and B Lymphocyte Homeostasis

5. Human TCR α/β+CD4−CD8−Double-Negative T Cells in Patients with Autoimmune Lymphoproliferative Syndrome Express Restricted Vβ TCR Diversity and Are Clonally Related to CD8+T Cells

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