NLRP12 is an innate immune checkpoint for repressing IFN signatures and attenuating lupus nephritis progression-Reference-Cited by-同舟云学术

NLRP12 is an innate immune checkpoint for repressing IFN signatures and attenuating lupus nephritis progression

Published:2023-02-01 Issue:3 Volume:133 Page:
ISSN:1558-8238
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Tsao Yen-Po^ORCID,Tseng Fang-Yu,Chao Chih-Wei,Chen Ming-Han^ORCID,Yeh Yi-Chen,Abdulkareem Babamale Olarewaju,Chen Se-Yi^ORCID,Chuang Wen-Ting,Chang Pei-Ching^ORCID,Chen I-Chun,Wang Pin-Hsuan,Wu Chien-Sheng^ORCID,Tsai Chang-Youh^ORCID,Chen Szu-Ting^ORCID

Funder

Ministry of Science and Technology, Taiwan

Yen Tjing Ling Medical Foundation

National Yang-Ming University Far Eastern Memorial Hospital Joint Research Program

National Yang Ming Chiao Tung University School of Medicine of Development and Construction program

Szu-Yuan Research Foundation of Internal Medicine

Cancer Progression Research Center, National Yang-Ming University, from the Higher Education Sprout Project by the Ministry of Education (MOE) in Taiwan

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://www.jci.org/articles/view/157272/files/pdf

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2. Nucleic acids of mammalian origin can act as endogenous ligands for Toll-like receptors and may promote systemic lupus erythematosus

3. Apoptosis-derived membrane vesicles drive the cGAS–STING pathway and enhance type I IFN production in systemic lupus erythematosus

4. TLR7 and TLR9 in SLE: when sensing self goes wrong

5. Autoreactive B Cell Responses to RNA-Related Antigens Due to TLR7 Gene Duplication

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