Cutting Edge: Th1 Cells Facilitate the Entry of Th17 Cells to the Central Nervous System during Experimental Autoimmune Encephalomyelitis-Reference-Cited by-同舟云学术

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Cutting Edge: Th1 Cells Facilitate the Entry of Th17 Cells to the Central Nervous System during Experimental Autoimmune Encephalomyelitis

Published:2008-09-03 Issue:6 Volume:181 Page:3750-3754
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

O'Connor Richard A.,Prendergast Catriona T.,Sabatos Catherine A.,Lau Clement W. Z.,Leech Melanie D.,Wraith David C.,Anderton Stephen M.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference16 articles.

1. Inflammatory leukocytes and cytokines in the peptide-induced disease of experimental allergic encephalomyelitis in SJL and B10.PL mice.

2. Encephalitogenic T cells in the B10.PL model of experimental allergic encephalomyelitis (EAE) are of the Th-1 lymphokine subtype

3. Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain

4. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

5. IL-25 regulates Th17 function in autoimmune inflammation

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