Th17 cell promotes apoptosis of IL-23R+ neurons in experimental autoimmune encephalomyelitis-Reference-Cited by-同舟云学术

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Th17 cell promotes apoptosis of IL-23R+ neurons in experimental autoimmune encephalomyelitis

Published:2024-02 Issue: Volume:259 Page:109898
ISSN:1521-6616
Container-title:Clinical Immunology
language:en
Short-container-title:Clinical Immunology

Author:

Sonar Sandip Ashok,Meitei Heikrujam Thoihen,Karmakar Surojit,Mishra Amrita,Inamdar Shrirang,Lenka Nibedita,Lal Girdhari

Funder

Department of Science and Technology, Ministry of Science and Technology, India

Science and Engineering Research Board

Ministry of Science and Technology

National Centre for Cell Science

Department of Biotechnology, Ministry of Science and Technology, India

Publisher

Elsevier BV

Reference54 articles.

1. T-bet regulates the fate of Th1 and Th17 lymphocytes in autoimmunity;Gocke;J. Immunol.,2007

2. The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells;Ivanov;Cell,2006

3. The transcription factors T-bet and Runx are required for the ontogeny of pathogenic interferon-gamma-producing T helper 17 cells;Wang;Immunity,2014

4. T-bet is essential for encephalitogenicity of both Th1 and Th17 cells;Yang;J. Exp. Med.,2009

5. Loss of T-bet, but not STAT1, prevents the development of experimental autoimmune encephalomyelitis;Bettelli;J. Exp. Med.,2004

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