IL-22 Is Expressed by Th17 Cells in an IL-23-Dependent Fashion, but Not Required for the Development of Autoimmune Encephalomyelitis-Reference-Cited by-同舟云学术

IL-22 Is Expressed by Th17 Cells in an IL-23-Dependent Fashion, but Not Required for the Development of Autoimmune Encephalomyelitis

Published:2007-12-03 Issue:12 Volume:179 Page:8098-8104
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Kreymborg Katharina,Etzensperger Ruth,Dumoutier Laure,Haak Stefan,Rebollo Angelita,Buch Thorsten,Heppner Frank L.,Renauld Jean-Christophe,Becher Burkhard

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference23 articles.

1. IL-12p35-Deficient Mice Are Susceptible to Experimental Autoimmune Encephalomyelitis: Evidence for Redundancy in the IL-12 System in the Induction of Central Nervous System Autoimmune Demyelination

2. Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain

3. Interleukin 18–independent engagement of interleukin 18 receptor-α is required for autoimmune inflammation

4. APC-derived cytokines and T cell polarization in autoimmune inflammation

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