Genome-Wide Expression Profiling Identifies an Impairment of Negative Feedback Signals in the Crohn’s Disease-Associated NOD2 Variant L1007fsinsC-Reference-Cited by-同舟云学术

Genome-Wide Expression Profiling Identifies an Impairment of Negative Feedback Signals in the Crohn’s Disease-Associated NOD2 Variant L1007fsinsC

Published:2011-02-18 Issue:7 Volume:186 Page:4027-4038
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J.I.

Author:

Billmann-Born Susanne,Till Andreas,Arlt Alexander,Lipinski Simone,Sina Christian,Latiano Anna,Annese Vito,Häsler Robert,Kerick Martin,Manke Thomas,Seegert Dirk,Hanidu Adedayo,Schäfer Heiner,van Heel David,Li Jun,Schreiber Stefan,Rosenstiel Philip

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference53 articles.

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3. Nod2 Is a General Sensor of Peptidoglycan through Muramyl Dipeptide (MDP) Detection

4. Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease

5. A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease

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