A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease-Reference-Cited by-同舟云学术

A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease

Published:2001-05 Issue:6837 Volume:411 Page:603-606
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Ogura Yasunori,Bonen Denise K.,Inohara Naohiro,Nicolae Dan L.,Chen Felicia F.,Ramos Richard,Britton Heidi,Moran Thomas,Karaliuskas Reda,Duerr Richard H.,Achkar Jean-Paul,Brant Steven R.,Bayless Theodore M.,Kirschner Barbara S.,Hanauer Stephen B.,Nuñez Gabriel,Cho Judy H.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/35079114.pdf

Reference22 articles.

1. Hugot, J. P. et al. Mapping of a susceptibility locus for Crohn's disease on chromosome 16. Nature 379, 821–823 (1996).

2. Ohmen, J. D. et al. Susceptibility locus for inflammatory bowel disease on chromosome 16 has a role in Crohn's disease, but not in ulcerative colitis. Hum. Mol. Genet. 5, 1679–1683 (1996).

3. Curran, M. E. et al. Genetic analysis of inflammatory bowel disease in a large European cohort supports linkage to chromosomes 12 and 16. Gastroenterology 115, 1066–1071 (1998).

4. Cavanaugh, J. A. et al. Analysis of Australian Crohn's disease pedigrees refines the localization for susceptibility to inflammatory bowel disease on chromosome 16. Ann. Hum. Genet. 62, 291–298 (1998).

5. Cho, J. H. et al. Identification of novel susceptibility loci for inflammatory bowel disease on chromosomes1p, 3q, and 4q: evidence for epistasis between 1p and IBD1. Proc. Natl Acad. Sci. USA 95, 7502–7507 (1998).

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