IL-6 Produced by Kupffer Cells Induces STAT Protein Activation in Hepatocytes Early During the Course of Systemic Listerial Infections
Author:
Affiliation:
1. *Department of Medicine and the
2. ‡Department of Cell Biology, Free University, Amsterdam, The Netherlands
3. †University of Pittsburgh Cancer Institute, University of Pittsburgh School of Medicine, Pittsburgh, PA 15213; and
Abstract
Publisher
The American Association of Immunologists
Subject
Immunology,Immunology and Allergy
Link
https://journals.aai.org/jimmunol/article-pdf/160/12/6056/1085347/im129806056o.pdf
Reference51 articles.
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2. Feder, L. S., J. A. Todaro, D. L. Laskin. 1993. Characterization of interleukin-1 and interleukin-6 production by hepatic endothelial cells and macrophages. J. Leukocyte Biol. 52: 126
3. Gregory, S. H., E. J. Wing. 1990. Accessory function of Kupffer cells in the antigen-specific blastogenic response of an L3T4+ T lymphocyte clone to Listeria monocytogenes. Infect. Immun. 58: 2313
4. Arnold, R., J. Scheffer, B. Konig, W. Konig. 1993. Effects of Listeria monocytogenes and Yersinia enterocolitica on cytokine gene expression and release from human polymorphonuclear granulocytes and epithelial (HEp-2) cells. Infect. Immun. 61: 2545
5. Xing, Z., M. Jordana, H. Kirpalani, K. E. Driscoll, T. J. Schall, J. Gauldie. 1994. Cytokine expression by neutrophils and macrophages in vivo: endotoxin induces tumor necrosis factor-alpha, macrophage inflammatory protein-2, interleukin-1β, and interleukin-6 but not RANTES or transforming growth factor-β1 mRNA expression in acute lung inflammation. Am. J. Respir. Cell Mol. Biol. 10: 148
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