CD8αβ T Cells Are Not Essential to the Pathogenesis of Arthritis or Colitis in HLA-B27 Transgenic Rats-Reference-Cited by-同舟云学术

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CD8αβ T Cells Are Not Essential to the Pathogenesis of Arthritis or Colitis in HLA-B27 Transgenic Rats

Published:2003-01-15 Issue:2 Volume:170 Page:1099-1105
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

May Ekkehard,Dorris Martha L.,Satumtira Nimman,Iqbal Imran,Rehman Muhammad I.,Lightfoot Ellis,Taurog Joel D.

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference45 articles.

1. Update on Spondyloarthropathies

2. Guilt by association: HLA-B27 and ankylosing spondylitis

3. HLA-B27-restricted CD8 T cells derived from synovial fluids of patients with reactive arthritis and ankylosing spondylitis

4. Minimal alterations in the HLA-B27-bound peptide repertoire induced upon infection of lymphoid cells withSalmonella typhimurium

5. The Peptide Repertoires of HLA-B27 Subtypes Differentially Associated to Spondyloarthropathy (B*2704 and B*2706) Differ by Specific Changes at Three Anchor Positions

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1. Emerging biochemical, microbial and immunological evidence in the search for why HLA-B∗27 confers risk for spondyloarthritis;Cell Chemical Biology;2024-08

2. CHOP-mediated IL-23 overexpression does not drive colitis in experimental spondyloarthritis;Scientific Reports;2024-05-29

3. Single-cell transcriptome revealed dysregulated RNA-binding protein expression patterns and functions in human ankylosing spondylitis;Frontiers in Medicine;2024-05-06

4. Spondyloarthritis;The Rose and Mackay Textbook of Autoimmune Diseases;2024

5. Vesicular traffic-mediated cell-to-cell signaling at the immune synapse in Ankylosing Spondylitis;Frontiers in Immunology;2023-01-18

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