IL-13 Regulates the Immune Response to Inhaled Antigens-Reference-Cited by-同舟云学术

IL-13 Regulates the Immune Response to Inhaled Antigens

Published:2005-06-08 Issue:12 Volume:174 Page:8097-8105
ISSN:0022-1767
Container-title:The Journal of Immunology
language:en
Short-container-title:J Immunol

Author:

Padilla Jocelyn,Daley Eleen,Chow Anthony,Robinson Kesha,Parthasarathi Kaushik,McKenzie Andrew N. J.,Tschernig Thomas,Kurup Viswanath P.,Donaldson Debra D.,Grunig Gabriele

Publisher

The American Association of Immunologists

Subject

Immunology,Immunology and Allergy

Reference61 articles.

1. Interleukin-13: Central Mediator of Allergic Asthma

2. Requirement for IL-13 Independently of IL-4 in Experimental Asthma

3. Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production

4. Interleukin-13 Mediates a Fundamental Pathway for Airway Epithelial Mucus Induced by CD4 T Cells and Interleukin-9

5. Direct effects of interleukin-13 on epithelial cells cause airway hyperreactivity and mucus overproduction in asthma

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