Cardiac troponin T mutations result in allele-specific phenotypes in a mouse model for hypertrophic cardiomyopathy-Reference-Cited by-同舟云学术

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Cardiac troponin T mutations result in allele-specific phenotypes in a mouse model for hypertrophic cardiomyopathy

Published:1999-08-15 Issue:4 Volume:104 Page:469-481
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Tardiff Jil C.,Hewett Timothy E.,Palmer Bradley M.,Olsson Charlotte,Factor Stephen M.,Moore Russell L.,Robbins Jeffrey,Leinwand Leslie A.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference43 articles.

1. The role of cytoskeletal proteins in cardiomyopathies

2. Familial hypertrophic cardiomyopathy: a genetic model of cardiac hypertrophy

3. Heterologous expression of a cardiomyopathic myosin that is defective in its actin interaction.

4. Functional analysis of the mutations in the human cardiac beta-myosin that are responsible for familial hypertrophic cardiomyopathy. Implication for the clinical outcome.

5. Functional Analysis of Myosin Mutations That Cause Familial Hypertrophic Cardiomyopathy

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1. Substrate mechanics unveil early structural and functional pathology in iPSC micro-tissue models of hypertrophic cardiomyopathy;iScience;2024-06

2. Modeling cardiomyocyte signaling and metabolism predicts genotype-to-phenotype mechanisms in hypertrophic cardiomyopathy;Computers in Biology and Medicine;2024-06

3. Allele-specific dysregulation of lipid and energy metabolism in early-stage hypertrophic cardiomyopathy;Journal of Molecular and Cellular Cardiology Plus;2024-06

4. Single-nucleus RNA/ATAC-seq in early-stage HCM models predicts SWI/SNF-activation in mutant-myocytes, and allele-specific differences in fibroblasts;2024-04-24

5. An In Silico Cardiomyocyte Reveals the Impact of Changes in CaMKII Signalling on Cardiomyocyte Contraction Kinetics in Hypertrophic Cardiomyopathy;BioMed Research International;2024-03-25

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