Heterologous expression of a cardiomyopathic myosin that is defective in its actin interaction.
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference25 articles.
1. A molecular basis for familial hypertrophic cardiomyopathy: A β cardiac myosin heavy chain gene missense mutation
2. Characteristics and Prognostic Implications of Myosin Missense Mutations in Familial Hypertrophic Cardiomyopathy
3. Differences in clinical expression of hypertrophic cardiomyopathy associated with two distinct mutations in the beta-myosin heavy chain gene. A 908Leu----Val mutation and a 403Arg----Gln mutation.
4. Substructure of the myosin molecule
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2. Virus-free transfection, transient expression, and purification of human cardiac myosin in mammalian muscle cells for biochemical and biophysical assays;Scientific Reports;2023-03-12
3. From Genetic Mutations to Molecular Basis of Heart Failure Treatment: An Overview of the Mechanism and Implication of the Novel Modulators for Cardiac Myosin;International Journal of Molecular Sciences;2021-06-21
4. Modeling Hypertrophic Cardiomyopathy: Mechanistic Insights and Pharmacological Intervention;Trends in Molecular Medicine;2019-09
5. All-Atom Molecular Dynamics Simulations of Actin–Myosin Interactions: A Comparative Study of Cardiac α Myosin, β Myosin, and Fast Skeletal Muscle Myosin;Biochemistry;2013-11-13
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