A dominant role for glucose in β cell compensation of insulin resistance
Author:
Publisher
American Society for Clinical Investigation
Subject
General Medicine
Reference21 articles.
1. Development of a Novel Polygenic Model of NIDDM in Mice Heterozygous for IR and IRS-1 Null Alleles
2. β-Cell Deficit and Increased β-Cell Apoptosis in Humans With Type 2 Diabetes
3. Relationship Between β-Cell Mass and Fasting Blood Glucose Concentration in Humans
4. Perspective: Postnatal Pancreatic β Cell Growth
5. Glucokinase and IRS-2 are required for compensatory cell hyperplasia in response to high-fat diet-induced insulin resistance
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