Glucokinase as a therapeutic target based on findings from the analysis of mouse models
Author:
Affiliation:
1. Department of Rheumatology, Endocrinology and Nephrology, Faculty of Medicine and Graduate School of Medicine, Hokkaido University, Sapporo, Japan
Publisher
Japan Endocrine Society
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
https://www.jstage.jst.go.jp/article/endocrj/69/5/69_EJ21-0742/_pdf
Reference54 articles.
1. 1 Kahn SE (2003) The relative contributions of insulin resistance and β-cell dysfunction to the pathophysiology of type 2 diabetes. Diabetologia 46: 3–19.
2. 2 Morimoto A, Tatsumi Y, Deura K, Mizuno S, Ohno Y, et al. (2013) Impact of impaired insulin secretion and insulin resistance on the incidence of type 2 diabetes mellitus in a Japanese population: the Saku study. Diabetologia 56: 1671–1679.
3. 3 Ohn JH, Kwak SH, Cho YM, Lim S, Jang HC, et al. (2016) 10-year trajectory of beta-cell function and insulin sensitivity in the development of type 2 diabetes: a community-based prospective cohort study. Lancet Diabetes Endocrinol 4: 27–34.
4. 4 Fujikawa R, Ito C, Kira S, Misumi M (2020) Longitudinal examination of pancreatic β-cell function in Japanese individuals. J Diabetes Investig 11: 70–74.
5. 5 Butler AE, Janson J, Bonner-Weir S, Ritzel R, Rizza RA, et al. (2003) β-cell deficit and increased beta-cell apoptosis in humans with type 2 diabetes. Diabetes 52: 102–110.
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