STRADα deficiency results in aberrant mTORC1 signaling during corticogenesis in humans and mice-Reference-Cited by-同舟云学术

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STRADα deficiency results in aberrant mTORC1 signaling during corticogenesis in humans and mice

Published:2010-05-03 Issue:5 Volume:120 Page:1591-1602
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:J. Clin. Invest.

Author:

Orlova Ksenia A.,Parker Whitney E.,Heuer Gregory G.,Tsai Victoria,Yoon Jason,Baybis Marianna,Fenning Robert S.,Strauss Kevin,Crino Peter B.

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Reference52 articles.

1. Polyhydramnios, megalencephaly and symptomatic epilepsy caused by a homozygous 7-kilobase deletion in LYK5

2. ATP and MO25α Regulate the Conformational State of the STRADα Pseudokinase and Activation of the LKB1 Tumour Suppressor

3. Activation of the tumour suppressor kinase LKB1 by the STE20-like pseudokinase STRAD

4. STRADα Regulates LKB1 Localization by Blocking Access to Importin-α, and by Association with Crm1 and Exportin-7

5. MO25 / interact with STRAD / enhancing their ability to bind, activate and localize LKB1 in the cytoplasm

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