Mitochondria-dependent ferroptosis plays a pivotal role in doxorubicin cardiotoxicity-Reference-Cited by-同舟云学术

Mitochondria-dependent ferroptosis plays a pivotal role in doxorubicin cardiotoxicity

Published:2020-05-07 Issue:9 Volume:5 Page:
ISSN:2379-3708
Container-title:JCI Insight
language:en
Short-container-title:

Author:

Tadokoro Tomonori,Ikeda Masataka^ORCID,Ide Tomomi,Deguchi Hiroko,Ikeda Soichiro,Okabe Kosuke,Ishikita Akihito,Matsushima Shouji,Koumura Tomoko,Yamada Ken-ichi^ORCID,Imai Hirotaka^ORCID,Tsutsui Hiroyuki

Funder

Japan Society for the Promotion of Science

Uehara Memorial Foundation

Takeda Science Foundation

Japan Agency for Medical Research and Development

Japan Foundation for Applied Enzymology

MSD Life Science Foundation, Public Interest Incorporated and Foundation

YOKOYAMA Foundation for Clinical Pharmacology

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://insight.jci.org/articles/view/132747/files/pdf

Reference55 articles.

1. Underlying Causes and Long-Term Survival in Patients with Initially Unexplained Cardiomyopathy

2. Doxorubicin-induced cardiomyopathy: From molecular mechanisms to therapeutic strategies

3. Identification of the molecular basis of doxorubicin-induced cardiotoxicity

4. CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress–induced myocardial necroptosis

5. Cardiotoxicity of doxorubicin is mediated through mitochondrial iron accumulation

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