Activation of SIRT3 by Tanshinone IIA ameliorates renal fibrosis by suppressing the TGF-β/TSP-1 pathway and attenuating oxidative stress-Reference-Cited by-同舟云学术

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Activation of SIRT3 by Tanshinone IIA ameliorates renal fibrosis by suppressing the TGF-β/TSP-1 pathway and attenuating oxidative stress

Published:2024-10 Issue: Volume:122 Page:111348
ISSN:0898-6568
Container-title:Cellular Signalling
language:en
Short-container-title:Cellular Signalling

Author:

Fan Yifeng,Kang Shengyu,Shao Tong,Xu Linhao,Chen Jian

Funder

Medical and Health Research Project of Zhejiang Province

Medical Technology and Education of Zhejiang Province of China

Department of Agriculture and Rural Affairs of Zhejiang Province

National Natural Science Foundation of China-Yunnan Joint Fund

Publisher

Elsevier BV

Reference53 articles.

1. Mitochondria-dependent ferroptosis plays a pivotal role in doxorubicin cardiotoxicity;Tadokoro;JCI Insight,2020

2. Protective effects of dioscin against doxorubicin-induced hepatotoxicity via regulation of Sirt1/FOXO1/NF-kappab signal;Song;Front. Pharmacol.,2019

3. Alleviation of the doxorubicin-induced nephrotoxicity by fasudil in vivo and in vitro;Xiang;J. Pharmacol. Sci.,2021

4. Protective effect of diosmin against doxorubicin-induced nephrotoxicity;Ali;Saudi J Biol Sci,2021

5. Protective effects of dioscin against doxorubicin-induced nephrotoxicity via adjusting FXR-mediated oxidative stress and inflammation;Zhang;Toxicology,2017

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